Výsledky vyhledávání - Elizabeth L. Tudor
- Zobrazuji výsledky 1 - 5 z 5
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1
The first ALS2 missense mutation associated with JPLS reveals new aspects of alsin biological function Autor Chris Panzeri, Clara De Palma, Andrea Martinuzzi, Andrea Daga, G. De Polo, Nereo Bresolin, Christopher C.J. Miller, Elizabeth L. Tudor, Emilio Clementi, Maria Teresa Bassi
Vydáno 2006Artigo -
2
VAPB interacts with the mitochondrial protein PTPIP51 to regulate calcium homeostasis Autor Kurt J. De Vos, Gábor M. Mórotz, Radu Stoica, Elizabeth L. Tudor, Kwok‐Fai Lau, Steven Ackerley, Alice Warley, Christopher E. Shaw, Christopher C.J. Miller
Vydáno 2011Artigo -
3
Overexpression of human wild-type FUS causes progressive motor neuron degeneration in an age- and dose-dependent fashion Autor Jacqueline C. Mitchell, Philip McGoldrick, Caroline Vance, Tibor Hortobágyi, Jemeen Sreedharan, Boris Rogelj, Elizabeth L. Tudor, Bradley Smith, Christian Klasen, Christopher C.J. Miller, Jonathan D. Cooper, Linda Greensmith, Christopher E. Shaw
Vydáno 2012Artigo -
4
ALS2/Alsin Regulates Rac-PAK Signaling and Neurite Outgrowth Autor Elizabeth L. Tudor, Michael S. Perkinton, Anja Schmidt, Steven Ackerley, Janet Brownlees, J.O. Nicholas Jacobsen, Helen L. Byers, Malcolm Ward, Alan Hall, P. Nigel Leigh, Christopher E. Shaw, Declan M. McLoughlin, Christopher C.J. Miller
Vydáno 2005Artigo -
5
Familial amyotrophic lateral sclerosis-linked SOD1 mutants perturb fast axonal transport to reduce axonal mitochondria content Autor Kurt J. De Vos, Anna Chapman, Maria E. Tennant, Catherine Manser, Elizabeth L. Tudor, Kwok‐Fai Lau, Janet Brownlees, Steven Ackerley, Pamela J. Shaw, Declan M. McLoughlin, Christopher E. Shaw, P. Nigel Leigh, Christopher C.J. Miller, Andrew J. Grierson
Vydáno 2007Artigo
Vyhledávací nástroje:
Související témata
Biology
Cell biology
Medicine
Amyotrophic lateral sclerosis
Disease
Gene
Pathology
Biochemistry
Genetics
Mitochondrion
Neuroscience
Axon
Axoplasmic transport
CDC42
Calcium
Calcium metabolism
Chemistry
Cytoplasm
Cytoplasmic inclusion
Dementia
Enzyme
Frontotemporal dementia
Frontotemporal lobar degeneration
GTP'
GTPase
Genetically modified mouse
Growth cone
Guanine nucleotide exchange factor
Hereditary spastic paraplegia
Homeostasis