Ohcanbohtosat - Dariusz Pytel

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  1. 1
    Tumor progression and the different faces of the PERK kinase

    Tumor progression and the different faces of the PERK kinase Dahkki Dariusz Pytel, Ireneusz Majsterek, J. Alan Diehl

    Almmustuhtton 2015
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  2. 2
    UPR-inducible miRNAs contribute to stressful situations

    UPR-inducible miRNAs contribute to stressful situations Dahkki Nilesh Chitnis, Dariusz Pytel, J. Alan Diehl

    Almmustuhtton 2013
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  3. 3
    Unfolded Protein Response and PERK Kinase as a New Therapeutic Target in the Pathogenesis of Alzheimer’s Disease

    Unfolded Protein Response and PERK Kinase as a New Therapeutic Target in the Pathogenesis of Alzheimer’s Disease Dahkki Wioletta Rozpędek‐Kamińska, Łukasz Markiewicz, Jean‐Luc Diehl, Dariusz Pytel, Ireneusz Majsterek

    Almmustuhtton 2015
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  4. 4
    The Role of the PERK/eIF2α/ATF4/CHOP Signaling Pathway in Tumor Progression During Endoplasmic Reticulum Stress

    The Role of the PERK/eIF2α/ATF4/CHOP Signaling Pathway in Tumor Progression During Endoplasmic Reticulum Stress Dahkki Wioletta Rozpędek‐Kamińska, Dariusz Pytel, Bartosz Mucha, H. Leszczynska, J. Alan Diehl, Ireneusz Majsterek

    Almmustuhtton 2016
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  5. 5
    The Structure, Activation and Signaling of IRE1 and Its Role in Determining Cell Fate

    The Structure, Activation and Signaling of IRE1 and Its Role in Determining Cell Fate Dahkki Natalia Siwecka, Wioletta Rozpędek‐Kamińska, Adam Wawrzynkiewicz, Dariusz Pytel, J. Alan Diehl, Ireneusz Majsterek

    Almmustuhtton 2021
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  6. 6
    Altered Expression Levels of MMP1, MMP9, MMP12, TIMP1, and IL-1<mml:math xmlns:mml="http://www.w3.org/1998/Math/MathML" id="M1"><mml:mrow><mml:mo mathvariant="bold">β</mml:mo></mml:mrow></mml:math>as a Risk Factor for the Elevated IOP and Optic Nerve Head Damage in the Primary Open-Angle Glaucoma Patients

    Altered Expression Levels of MMP1, MMP9, MMP12, TIMP1, and IL-1<mml:math xmlns:mml="http://www.w3.org/1998/Math/MathML" id="M1"><mml:mrow><mml:mo mathvariant="bold">β</mml:mo></mml... Dahkki Łukasz Markiewicz, Dariusz Pytel, Bartosz Mucha, Katarzyna Szymanek, Jerzy Szaflik, Jacek P. Szaflik, Ireneusz Majsterek

    Almmustuhtton 2015
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  7. 7
    The PERK-Dependent Molecular Mechanisms as a Novel Therapeutic Target for Neurodegenerative Diseases

    The PERK-Dependent Molecular Mechanisms as a Novel Therapeutic Target for Neurodegenerative Diseases Dahkki Wioletta Rozpędek‐Kamińska, Natalia Siwecka, Adam Wawrzynkiewicz, Radosław Wojtczak, Dariusz Pytel, J. Alan Diehl, Ireneusz Majsterek

    Almmustuhtton 2020
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  8. 8
    Dual role of Endoplasmic Reticulum Stress-Mediated Unfolded Protein Response Signaling Pathway in Carcinogenesis

    Dual role of Endoplasmic Reticulum Stress-Mediated Unfolded Protein Response Signaling Pathway in Carcinogenesis Dahkki Natalia Siwecka, Wioletta Rozpędek‐Kamińska, Dariusz Pytel, Adam Wawrzynkiewicz, Adam Dziki, Łukasz Dziki, J. Alan Diehl, Ireneusz Majsterek

    Almmustuhtton 2019
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  9. 9
    PERK promotes cancer cell proliferation and tumor growth by limiting oxidative DNA damage

    PERK promotes cancer cell proliferation and tumor growth by limiting oxidative DNA damage Dahkki Ekaterina Bobrovnikova-Marjon, Christina Grigoriadou, Dariusz Pytel, F Zhang, Jiangbin Ye, Constantinos Koumenis, Douglas R. Cavener, J. Alan Diehl

    Almmustuhtton 2010
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  10. 10
    PERK Utilizes Intrinsic Lipid Kinase Activity To Generate Phosphatidic Acid, Mediate Akt Activation, and Promote Adipocyte Differentiation

    PERK Utilizes Intrinsic Lipid Kinase Activity To Generate Phosphatidic Acid, Mediate Akt Activation, and Promote Adipocyte Differentiation Dahkki Ekaterina Bobrovnikova-Marjon, Dariusz Pytel, Matthew J. Riese, Laura Pontano Vaites, Nickpreet Singh, Gary A. Koretzky, Eric S. Witze, J. Alan Diehl

    Almmustuhtton 2012
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  11. 11
    BCR/ABL Stimulates WRN to Promote Survival and Genomic Instability

    BCR/ABL Stimulates WRN to Promote Survival and Genomic Instability Dahkki Artur Słupianek, Tomasz Popławski, Stanisław K. Jóźwiakowski, Kimberly Cramer, Dariusz Pytel, Ewelina Stoczyńska-Fidelus, Michal O. Nowicki, Janusz Błasiak, Tomasz Skórski

    Almmustuhtton 2010
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  12. 12
    BCR-ABL1 kinase inhibits uracil DNA glycosylase UNG2 to enhance oxidative DNA damage and stimulate genomic instability

    BCR-ABL1 kinase inhibits uracil DNA glycosylase UNG2 to enhance oxidative DNA damage and stimulate genomic instability Dahkki Artur Słupianek, Rafal Falinski, Paweł Znojek, Tomasz Stokłosa, Sylwia Flis, Valentina Doneddu, Dariusz Pytel, Ewelina Synowiec, Janusz Błasiak, Alfonso Bellacosa, Tomasz Skórski

    Almmustuhtton 2012
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  13. 13
    The ζ Isoform of Diacylglycerol Kinase Plays a Predominant Role in Regulatory T Cell Development and TCR-Mediated Ras Signaling

    The ζ Isoform of Diacylglycerol Kinase Plays a Predominant Role in Regulatory T Cell Development and TCR-Mediated Ras Signaling Dahkki Rohan P. Joshi, Amanda M. Schmidt, Jayajit Das, Dariusz Pytel, Matthew J. Riese, Melissa T. Lester, J. Alan Diehl, Edward M. Behrens, Taku Kambayashi, Gary A. Koretzky

    Almmustuhtton 2013
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  14. 14
    miR-211 Is a Prosurvival MicroRNA that Regulates chop Expression in a PERK-Dependent Manner

    miR-211 Is a Prosurvival MicroRNA that Regulates chop Expression in a PERK-Dependent Manner Dahkki Nilesh Chitnis, Dariusz Pytel, Ekaterina Bobrovnikova-Marjon, Dhruv K. Pant, Hui Zheng, Nancy L. Maas, Brian Frederick, Jake A. Kushner, Lewis A. Chodosh, Constantinos Koumenis, Serge Y. Fuchs, J. Alan Diehl

    Almmustuhtton 2012
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  15. 15
    PERK Is a Haploinsufficient Tumor Suppressor: Gene Dose Determines Tumor-Suppressive Versus Tumor Promoting Properties of PERK in Melanoma

    PERK Is a Haploinsufficient Tumor Suppressor: Gene Dose Determines Tumor-Suppressive Versus Tumor Promoting Properties of PERK in Melanoma Dahkki Dariusz Pytel, Yan Gao, Katarzyna Mackiewicz, Yuliya V. Katlinskaya, Kirk A. Staschke, María Cristina Gavilanes Paredes, Akihiro Yoshida, Shuo Qie, Gao Zhang, Olga S. Chajewski, Lawrence W. Wu, Ireneusz Majsterek, Meenhard Herlyn, Serge Y. Fuchs, J. Alan Diehl

    Almmustuhtton 2016
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  16. 16
    Carbon Monoxide Activates PERK-Regulated Autophagy to Induce Immunometabolic Reprogramming and Boost Antitumor T-cell Function

    Carbon Monoxide Activates PERK-Regulated Autophagy to Induce Immunometabolic Reprogramming and Boost Antitumor T-cell Function Dahkki Paramita Chakraborty, Rasesh Y. Parikh, Sang Ho Choi, Danh T. Tran, Mónika Göőz, Zachariah Hedley, Do‐Sung Kim, Dariusz Pytel, In‐Hong Kang, Satish N. Nadig, Gyda C. Beeson, Lauren E. Ball, Meenal Mehrotra, Hongjun Wang, Stefano Berto, Viswanathan Palanisamy, Hong Li, Shilpak Chatterjee, Paulo C. Rodrı́guez, Eduardo N. Maldonado, J. Alan Diehl, Vamsi K. Gangaraju, Shikhar Mehrotra

    Almmustuhtton 2022
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