检索结果 - Ching‐Hwa Tsai

  • Showing 1 - 17 results of 17
Refine Results
  1. 1
    Epstein-Barr Virus Latent Membrane Protein 2A Regulates c-Jun Protein through Extracellular Signal-Regulated Kinase

    Epstein-Barr Virus Latent Membrane Protein 2A Regulates c-Jun Protein through Extracellular Signal-Regulated Kinase 由 Shaoyin Chen, Jean Lu, Yin-Chu Shih, Ching-Hwa Tsai

    出版 2002
    获取全文
    Artigo
    加到收藏夹
    Saved in:
  2. 2
    Requirement for Cell-to-Cell Contact in Epstein-Barr Virus Infection of Nasopharyngeal Carcinoma Cells and Keratinocytes

    Requirement for Cell-to-Cell Contact in Epstein-Barr Virus Infection of Nasopharyngeal Carcinoma Cells and Keratinocytes 由 Yao Chang, Che‐Huang Tung, Yu-Tzu Huang, Jean Lu, Jen‐Yang Chen, Ching-Hwa Tsai

    出版 1999
    获取全文
    Artigo
    加到收藏夹
    Saved in:
  3. 3
    Epstein-Barr Virus BGLF4 Kinase Suppresses the Interferon Regulatory Factor 3 Signaling Pathway

    Epstein-Barr Virus BGLF4 Kinase Suppresses the Interferon Regulatory Factor 3 Signaling Pathway 由 Jiin-Tarng Wang, Shin-Lian Doong, Shu‐Chun Teng, Chung‐Pei Lee, Ching-Hwa Tsai, Mei‐Ru Chen

    出版 2008
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  4. 4
    Epstein-Barr-Virus-Encoded LMP2A Induces Primary Epithelial Cell Migration and Invasion: Possible Role in Nasopharyngeal Carcinoma Metastasis

    Epstein-Barr-Virus-Encoded LMP2A Induces Primary Epithelial Cell Migration and Invasion: Possible Role in Nasopharyngeal Carcinoma Metastasis 由 D. Michiel Pegtel, Aravind Subramanian, Tzung-Shiahn Sheen, Ching‐Hwa Tsai, Todd R. Golub, David A. Thorley‐Lawson

    出版 2005
    获取全文
    Artigo
    加到收藏夹
    Saved in:
  5. 5
    Syk Tyrosine Kinase Mediates Epstein-Barr Virus Latent Membrane Protein 2A-induced Cell Migration in Epithelial Cells

    Syk Tyrosine Kinase Mediates Epstein-Barr Virus Latent Membrane Protein 2A-induced Cell Migration in Epithelial Cells 由 Jean Lu, Wan Hsin Lin, Shao-Yin Chen, Richard Longnecker, Shu-Chun Tsai, Chi-Long Chen, Ching-Hwa Tsai

    出版 2006
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  6. 6
    EBV Zta protein induces the expression of interleukin-13, promoting the proliferation of EBV-infected B cells and lymphoblastoid cell lines

    EBV Zta protein induces the expression of interleukin-13, promoting the proliferation of EBV-infected B cells and lymphoblastoid cell lines 由 Shu‐Chun Tsai, Sue‐Jane Lin, Po‐Wen Chen, Wen-Yi Luo, Te‐Huei Yeh, Hsei-Wei Wang, Chi‐Ju Chen, Ching-Hwa Tsai

    出版 2009
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  7. 7
    Epstein-Barr virus LMP2A suppresses MHC class II expression by regulating the B-cell transcription factors E47 and PU.1

    Epstein-Barr virus LMP2A suppresses MHC class II expression by regulating the B-cell transcription factors E47 and PU.1 由 Jiun-Han Lin, Ju-Yin Lin, Ya‐Ching Chou, Mei‐Ru Chen, Te‐Huei Yeh, Chung‐Wu Lin, Sue-Jane Lin, Ching‐Hwa Tsai

    出版 2015
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  8. 8
    Inhibition of Epstein-Barr virus reactivation by the flavonoid apigenin

    Inhibition of Epstein-Barr virus reactivation by the flavonoid apigenin 由 Chung-Chun Wu, Chih‐Yeu Fang, Yu-Jhen Cheng, Hui‐Yu Hsu, Sheng‐Ping Chou, Sheng‐Yen Huang, Ching-Hwa Tsai, Jen‐Yang Chen

    出版 2017
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  9. 9
    Epstein-Barr Virus Lytic Transactivator Zta Enhances Chemotactic Activity through Induction of Interleukin-8 in Nasopharyngeal Carcinoma Cells

    Epstein-Barr Virus Lytic Transactivator Zta Enhances Chemotactic Activity through Induction of Interleukin-8 in Nasopharyngeal Carcinoma Cells 由 Mei‐Chi Hsu, Shih-Yi Wu, Shih-Shin Chang, Ih‐Jen Su, Ching-Hwa Tsai, Siao-Jing Lai, Ai‐Li Shiau, Kenzo Takada, Yao Chang

    出版 2008
    获取全文
    Artigo
    加到收藏夹
    Saved in:
  10. 10
    CTL Control of EBV in Nasopharyngeal Carcinoma (NPC): EBV-Specific CTL Responses in the Blood and Tumors of NPC Patients and the Antigen-Processing Function of the Tumor Cells

    CTL Control of EBV in Nasopharyngeal Carcinoma (NPC): EBV-Specific CTL Responses in the Blood and Tumors of NPC Patients and the Antigen-Processing Function of the Tumor Cells 由 Steven P. Lee, Anthony T.�C. Chan, ST Cheung, Wendy A. Thomas, Debbie Croom-Carter, Chris Dawson, Ching‐Hwa Tsai, Sing-Fai Leung, Philip J. Johnson, Dolly P. Huang

    出版 2000
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  11. 11
    Deoxycytidine deaminase-resistant stereoisomer is the active form of (+/-)-2',3'-dideoxy-3'-thiacytidine in the inhibition of hepatitis B virus replication.

    Deoxycytidine deaminase-resistant stereoisomer is the active form of (+/-)-2',3'-dideoxy-3'-thiacytidine in the inhibition of hepatitis B virus replication. 由 C.N. Chang, Shin-Lian Doong, Junhu Zhou, Joseph Warren Beach, Lak Shin Jeong, C. K. CHU, Ching-hwa Tsai, Ying‐Chih Cheng, Dennis C. Liotta, Raymond F. Schinazi

    出版 1992
    获取全文
    Artigo
    加到收藏夹
    Saved in:
  12. 12
    Epstein-Barr Virus BGLF4 Kinase Retards Cellular S-Phase Progression and Induces Chromosomal Abnormality

    Epstein-Barr Virus BGLF4 Kinase Retards Cellular S-Phase Progression and Induces Chromosomal Abnormality 由 Yu-Hsin Chang, Chung‐Pei Lee, Mei-Tzu Su, Jiin-Tarng Wang, Jen‐Yang Chen, Su‐Fang Lin, Ching‐Hwa Tsai, Min-Jei Hsieh, Kenzo Takada, Mei‐Ru Chen

    出版 2012
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  13. 13
    EGCG Inhibits Proliferation, Invasiveness and Tumor Growth by Up-Regulation of Adhesion Molecules, Suppression of Gelatinases Activity, and Induction of Apoptosis in Nasopharyngeal Carcinoma Cells

    EGCG Inhibits Proliferation, Invasiveness and Tumor Growth by Up-Regulation of Adhesion Molecules, Suppression of Gelatinases Activity, and Induction of Apoptosis in Nasopharyngeal... 由 Chih‐Yeu Fang, Chung-Chun Wu, Hui‐Yu Hsu, Hsin-Ying Chuang, Sheng-Yen Huang, Ching‐Hwa Tsai, Yao Chang, George Sai‐Wah Tsao, Chi‐Long Chen, Jen‐Yang Chen

    出版 2015
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  14. 14
    The ESCRT Machinery Is Recruited by the Viral BFRF1 Protein to the Nucleus-Associated Membrane for the Maturation of Epstein-Barr Virus

    The ESCRT Machinery Is Recruited by the Viral BFRF1 Protein to the Nucleus-Associated Membrane for the Maturation of Epstein-Barr Virus 由 Chung-Pei Lee, Po-Ting Liu, Hsiu‐Ni Kung, Mei-Tzu Su, Huey-Huey Chua, Yu-Hsin Chang, Chou-Wei Chang, Ching‐Hwa Tsai, Fu‐Tong Liu, Mei‐Ru Chen

    出版 2012
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  15. 15
    The pathological effects of CCR2+ inflammatory monocytes are amplified by an IFNAR1-triggered chemokine feedback loop in highly pathogenic influenza infection

    The pathological effects of CCR2+ inflammatory monocytes are amplified by an IFNAR1-triggered chemokine feedback loop in highly pathogenic influenza infection 由 Sue-Jane Lin, Ming Lo, Rei-Lin Kuo, Shin‐Ru Shih, David M. Ojcius, Jean Lu, Chien‐Kuo Lee, Hui‐Chen Chen, Meei Yun Lin, Chuen-Miin Leu, Chia‐Ni Lin, Ching‐Hwa Tsai

    出版 2014
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  16. 16
    Recurrent chemical reactivations of EBV promotes genome instability and enhances tumor progression of nasopharyngeal carcinoma cells

    Recurrent chemical reactivations of EBV promotes genome instability and enhances tumor progression of nasopharyngeal carcinoma cells 由 Chih‐Yeu Fang, Chia‐Huei Lee, Chung‐Chun Wu, Yu‐Ting Chang, Shu‐Ling Yu, Sheng‐Ping Chou, P J Huang, Chi‐Long Chen, Jia‐Woei Hou, Yao Chang, Ching‐Hwa Tsai, Kenzo Takada, Jen‐Yang Chen

    出版 2008
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:
  17. 17
    The Ubiquitin Ligase Itch and Ubiquitination Regulate BFRF1-Mediated Nuclear Envelope Modification for Epstein-Barr Virus Maturation

    The Ubiquitin Ligase Itch and Ubiquitination Regulate BFRF1-Mediated Nuclear Envelope Modification for Epstein-Barr Virus Maturation 由 Chung‐Pei Lee, Guan‐Ting Liu, Hsiu‐Ni Kung, Po-Ting Liu, Yen‐Tzu Liao, Lu‐Ping Chow, Ling-Shih Chang, Yu-Hsien Chang, Chou-Wei Chang, Wen-Chi Shu, Annie Angers, Antonella Farina, Su‐Fang Lin, Ching-Hwa Tsai, Fadila Bouamr, Mei‐Ru Chen

    出版 2016
    获取全文 获取全文
    Artigo
    加到收藏夹
    Saved in:

检索工具: