نتائج البحث - Chiaki T. Ishida

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  1. 1
    Targeting intrinsic apoptosis and other forms of cell death by BH3-mimetics in glioblastoma

    Targeting intrinsic apoptosis and other forms of cell death by BH3-mimetics in glioblastoma حسب Georg Karpel‐Massler, Chiaki Tsuge Ishida, Yiru Zhang, Marc‐Eric Halatsch, Mike‐Andrew Westhoff, Markus D. Siegelin

    منشور في 2017
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    Revisão
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  2. 2
    BH3-mimetics and BET-inhibitors elicit enhanced lethality in malignant glioma

    BH3-mimetics and BET-inhibitors elicit enhanced lethality in malignant glioma حسب Chiaki Tsuge Ishida, Elena Bianchetti, Chang Shu, Marc‐Eric Halatsch, Mike‐Andrew Westhoff, Georg Karpel‐Massler, Markus D. Siegelin

    منشور في 2017
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    Artigo
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  3. 3
    Induction of synthetic lethality in IDH1-mutated gliomas through inhibition of Bcl-xL

    Induction of synthetic lethality in IDH1-mutated gliomas through inhibition of Bcl-xL حسب Georg Karpel‐Massler, Chiaki Tsuge Ishida, Elena Bianchetti, Yiru Zhang, Chang Shu, Takashi Tsujiuchi, Matei A. Banu, Franklin Garcia, Kevin A. Roth, Jeffrey N. Bruce, P. Canoll, Markus D. Siegelin

    منشور في 2017
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    Artigo
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  4. 4
    Inhibition of Mitochondrial Matrix Chaperones and Antiapoptotic Bcl-2 Family Proteins Empower Antitumor Therapeutic Responses

    Inhibition of Mitochondrial Matrix Chaperones and Antiapoptotic Bcl-2 Family Proteins Empower Antitumor Therapeutic Responses حسب Georg Karpel‐Massler, Chiaki Tsuge Ishida, Elena Bianchetti, Chang Shu, Rolando Pérez‐Lorenzo, Basil A. Horst, Matei A. Banu, Kevin A. Roth, Jeffrey N. Bruce, Peter Canoll, Dario C. Altieri, Markus D. Siegelin

    منشور في 2017
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    Artigo
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  5. 5
    Metabolic Reprogramming by Dual AKT/ERK Inhibition through Imipridones Elicits Unique Vulnerabilities in Glioblastoma

    Metabolic Reprogramming by Dual AKT/ERK Inhibition through Imipridones Elicits Unique Vulnerabilities in Glioblastoma حسب Chiaki T. Ishida, Yiru Zhang, Elena Bianchetti, Chang Shu, Trang Nguyen, Giulio Kleiner, Maria J. Sanchez‐Quintero, Catarina M. Quinzii, Mike‐Andrew Westhoff, Georg Karpel‐Massler, Varun V. Prabhu, Joshua E. Allen, Markus D. Siegelin

    منشور في 2018
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    Artigo
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  6. 6
    Activation of <scp>LXR</scp> β inhibits tumor respiration and is synthetically lethal with Bcl‐ <scp>xL</scp> inhibition

    Activation of <scp>LXR</scp> β inhibits tumor respiration and is synthetically lethal with Bcl‐ <scp>xL</scp> inhibition حسب Trang Nguyen, Chiaki Tsuge Ishida, Enyuan Shang, Chang Shu, Consuelo Torrini, Yiru Zhang, Elena Bianchetti, Maria J. Sanchez‐Quintero, Giulio Kleiner, Catarina M. Quinzii, Mike‐Andrew Westhoff, Georg Karpel‐Massler, Peter Canoll, Markus D. Siegelin

    منشور في 2019
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    Artigo
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  7. 7
    Combined HDAC and Bromodomain Protein Inhibition Reprograms Tumor Cell Metabolism and Elicits Synthetic Lethality in Glioblastoma

    Combined HDAC and Bromodomain Protein Inhibition Reprograms Tumor Cell Metabolism and Elicits Synthetic Lethality in Glioblastoma حسب Yiru Zhang, Chiaki Tsuge Ishida, Wataru Ishida, Sheng-Fu Larry Lo, Junfei Zhao, Chang Shu, Elena Bianchetti, Giulio Kleiner, Maria J. Sanchez‐Quintero, Catarina M. Quinzii, Mike‐Andrew Westhoff, Georg Karpel‐Massler, Peter Canoll, Markus D. Siegelin

    منشور في 2018
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    Artigo
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