Hakutulokset - Chen, Jeng‐Haur

Protein kinase A phosphorylation potentiates cystic fibrosis transmembrane conductance regulator gating by relieving autoinhibition on the stimulatory C terminus of the regulatory... Tekijä Chen, Jeng-Haur

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Direct Sensing of Intracellular pH by the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) Cl(−) Channel Tekijä Chen, Jeng-Haur, Cai, Zhiwei, Sheppard, David N.

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Altering intracellular pH reveals the kinetic basis of intraburst gating in the CFTR Cl(−) channel Tekijä Chen, Jeng‐Haur, Xu, Weiyi, Sheppard, David N.

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Acute inhibition of the cystic fibrosis transmembrane conductance regulator (CFTR) Cl(−) channel by thyroid hormones involves multiple mechanisms Tekijä Cai, Zhiwei, Li, Hongyu, Chen, Jeng-Haur, Sheppard, David N.

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Human cystic fibrosis airway epithelia have reduced Cl(−) conductance but not increased Na(+) conductance Tekijä Itani, Omar A., Chen, Jeng-Haur, Karp, Philip H., Ernst, Sarah, Keshavjee, Shaf, Parekh, Kalpaj, Klesney-Tait, Julia, Zabner, Joseph, Welsh, Michael J.

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Inhibition of Protein Kinase CK2 Closes the CFTR Cl(−) Channel, but has no Effect on the Cystic Fibrosis Mutant ΔF508-CFTR Tekijä Treharne, Kate J., Xu, Zhe, Chen, Jeng-Haur, Best, O. Giles, Cassidy, Diane M., Gruenert, Dieter C., Hegyi, Péter, Gray, Michael A., Sheppard, David N., Kunzelmann, Karl, Mehta, Anil

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Protein kinase CK2, cystic fibrosis transmembrane conductance regulator, and the ΔF508 mutation. F508 deletion disrupts a kinase-binding site. VOLUME 282 (2007) PAGES 10804-10813 Tekijä Treharne, Kate J., Crawford, Russell M., Xu, Zhe, Chen, Jeng-Haur, Best, O. Giles, Schulte, Eva A., Gruenert, Dieter C., Wilson, Stuart M., Sheppard, David N., Kunzelmann, Kar, Mehta, Anil

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CFTR-deficient pigs display peripheral nervous system defects at birth Tekijä Reznikov, Leah R., Dong, Qian, Chen, Jeng-Haur, Moninger, Thomas O., Park, Jung Min, Zhang, Yuzhou, Du, Jianyang, Hildebrand, Michael S., Smith, Richard J. H., Randak, Christoph O., Stoltz, David A., Welsh, Michael J.

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Loss of anion transport without increased sodium absorption characterizes newborn porcine cystic fibrosis airway epithelia Tekijä Chen, Jeng-Haur, Stoltz, David A., Karp, Philip H., Ernst, Sarah E., Pezzulo, Alejandro A., Moninger, Thomas O., Rector, Michael V., Reznikov, Leah R., Launspach, Janice L., Chaloner, Kathryn, Zabner, Joseph, Welsh, Michael J.

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CFTR founder mutation causes protein trafficking defects in Chinese patients with cystic fibrosis Tekijä Leung, Gordon K. C., Ying, Dingge, Mak, Christopher C. Y., Chen, Xin‐Ying, Xu, Weiyi, Yeung, Kit‐San, Wong, Wai‐Lap, Chu, Yoyo W. Y., Mok, Gary T. K., Chau, Christy S. K., McLuskey, Jenna, Ong, Winnie P. T., Leong, Huey‐Yin, Chan, Kelvin Y. K., Yang, Wanling, Chen, Jeng‐Haur, Li, Albert M., Sham, Pak C., Lau, Yu‐Lung, Chung, Brian H. Y., Lee, So‐Lun

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The ΔF508 Mutation Causes CFTR Misprocessing and Cystic Fibrosis-Like Disease in Pigs Tekijä Ostedgaard, Lynda S, Meyerholz, David K, Chen, Jeng-Haur, Pezzulo, Alejandro A, Karp, Philip H, Rokhlina, Tatiana, Ernst, Sarah E, Hanfland, Robert A, Reznikov, Leah R, Ludwig, Paula S, Rogan, Mark P, Davis, Greg J, Dohrn, Cassie L, Wohlford-Lenane, Christine, Taft, Peter J, Rector, Michael V, Hornick, Emma, Nassar, Boulos S, Samuel, Melissa, Zhang, Yuping, Richter, Sandra S, Uc, Aliye, Shilyansky, Joel, Prather, Randall S, McCray, Paul B, Zabner, Joseph, Welsh, Michael J, Stoltz, David A

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Intestinal CFTR expression alleviates meconium ileus in cystic fibrosis pigs Tekijä Stoltz, David A., Rokhlina, Tatiana, Ernst, Sarah E., Pezzulo, Alejandro A., Ostedgaard, Lynda S., Karp, Philip H., Samuel, Melissa S., Reznikov, Leah R., Rector, Michael V., Gansemer, Nicholas D., Bouzek, Drake C., Alaiwa, Mahmoud H. Abou, Hoegger, Mark J., Ludwig, Paula S., Taft, Peter J., Wallen, Tanner J., Wohlford-Lenane, Christine, McMenimen, James D., Chen, Jeng-Haur, Bogan, Katrina L., Adam, Ryan J., Hornick, Emma E., Nelson, George A., Hoffman, Eric A., Chang, Eugene H., Zabner, Joseph, McCray, Paul B., Prather, Randall S., Meyerholz, David K., Welsh, Michael J.

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