Ngā hua rapu - Carol Feghali‐Bostwick

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  1. 1
    The Molecular Mechanisms of Systemic Sclerosis-Associated Lung Fibrosis

    The Molecular Mechanisms of Systemic Sclerosis-Associated Lung Fibrosis mā Joe E. Mouawad, Carol Feghali‐Bostwick

    I whakaputaina 2023
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Revisão
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  2. 2
    The Pro-Fibrotic Factor IGFBP-5 Induces Lung Fibroblast and Mononuclear Cell Migration

    The Pro-Fibrotic Factor IGFBP-5 Induces Lung Fibroblast and Mononuclear Cell Migration mā Hidekata Yasuoka, Yukie Yamaguchi, Carol Feghali‐Bostwick

    I whakaputaina 2009
    Whiwhi kuputuhi katoa
    Artigo
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  3. 3
    The Mighty Fibroblast and Its Utility in Scleroderma Research

    The Mighty Fibroblast and Its Utility in Scleroderma Research mā Sara M. Garrett, DeAnna Baker Frost, Carol Feghali‐Bostwick

    I whakaputaina 2017
    Whiwhi kuputuhi katoa
    Artigo
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  4. 4
    Interleukin‐13–producing CD8+ T cells mediate dermal fibrosis in patients with systemic sclerosis

    Interleukin‐13–producing CD8+ T cells mediate dermal fibrosis in patients with systemic sclerosis mā Patrizia Fuschiotti, Adriana T. Larregina, Johnan Ho, Carol Feghali‐Bostwick, Thomas A. Medsger

    I whakaputaina 2012
    Whiwhi kuputuhi katoa
    Artigo
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  5. 5
    Human lung myofibroblast TGFβ1-dependent Smad2/3 signalling is Ca2+-dependent and regulated by KCa3.1 K+ channels

    Human lung myofibroblast TGFβ1-dependent Smad2/3 signalling is Ca2+-dependent and regulated by KCa3.1 K+ channels mā Katy M. Roach, Carol Feghali‐Bostwick, Heike Wulff, Yassine Amrani, Peter Bradding

    I whakaputaina 2015
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  6. 6
    Repression of IP-10 by Interactions between Histone Deacetylation and Hypermethylation in Idiopathic Pulmonary Fibrosis

    Repression of IP-10 by Interactions between Histone Deacetylation and Hypermethylation in Idiopathic Pulmonary Fibrosis mā William R. Coward, Keira Watts, Carol Feghali‐Bostwick, Gisli Jenkins, Linhua Pang

    I whakaputaina 2010
    Whiwhi kuputuhi katoa
    Artigo
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  7. 7
    Increased constitutive αSMA and Smad2/3 expression in idiopathic pulmonary fibrosis myofibroblasts is KCa3.1-dependent

    Increased constitutive αSMA and Smad2/3 expression in idiopathic pulmonary fibrosis myofibroblasts is KCa3.1-dependent mā Katy M. Roach, Heike Wulff, Carol Feghali‐Bostwick, Yassine Amrani, Peter Bradding

    I whakaputaina 2014
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  8. 8
    IGFBP-5 Promotes Fibrosis via Increasing Its Own Expression and That of Other Pro-fibrotic Mediators

    IGFBP-5 Promotes Fibrosis via Increasing Its Own Expression and That of Other Pro-fibrotic Mediators mā Xinh-Xinh Nguyen, Lutfiyya N. Muhammad, Paul J. Nietert, Carol Feghali‐Bostwick

    I whakaputaina 2018
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  9. 9
    A central role for G9a and EZH2 in the epigenetic silencing of cyclooxygenase‐2 in idiopathic pulmonary fibrosis

    A central role for G9a and EZH2 in the epigenetic silencing of cyclooxygenase‐2 in idiopathic pulmonary fibrosis mā William R. Coward, Carol Feghali‐Bostwick, Gisli Jenkins, Alan J. Knox, Linhua Pang

    I whakaputaina 2014
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  10. 10
    Defective Histone Acetylation Is Responsible for the Diminished Expression of Cyclooxygenase 2 in Idiopathic Pulmonary Fibrosis

    Defective Histone Acetylation Is Responsible for the Diminished Expression of Cyclooxygenase 2 in Idiopathic Pulmonary Fibrosis mā William R. Coward, Keiria Watts, Carol Feghali‐Bostwick, Alan J. Knox, Linhua Pang

    I whakaputaina 2009
    Whiwhi kuputuhi katoa
    Artigo
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  11. 11
    Gender differences in systemic sclerosis: relationship to clinical features, serologic status and outcomes

    Gender differences in systemic sclerosis: relationship to clinical features, serologic status and outcomes mā Christine Peoples, Thomas A. Medsger, Mary Lucas, Bedda L. Rosario, Carol Feghali‐Bostwick

    I whakaputaina 2016
    Whiwhi kuputuhi katoa
    Artigo
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  12. 12
    Prominence of IL6, IGF, TLR, and Bioenergetics Pathway Perturbation in Lung Tissues of Scleroderma Patients With Pulmonary Fibrosis

    Prominence of IL6, IGF, TLR, and Bioenergetics Pathway Perturbation in Lung Tissues of Scleroderma Patients With Pulmonary Fibrosis mā Ludivine Renaud, Willian A. da Silveira, Naoko Takamura, Gary Hardiman, Carol Feghali‐Bostwick

    I whakaputaina 2020
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  13. 13
    Insulin-Like Growth Factor Binding Proteins 3 and 5 Are Overexpressed in Idiopathic Pulmonary Fibrosis and Contribute to Extracellular Matrix Deposition

    Insulin-Like Growth Factor Binding Proteins 3 and 5 Are Overexpressed in Idiopathic Pulmonary Fibrosis and Contribute to Extracellular Matrix Deposition mā Joseph M. Pilewski, Lixin Liu, Adam C. Henry, Alycia V. Knauer, Carol Feghali‐Bostwick

    I whakaputaina 2005
    Whiwhi kuputuhi katoa
    Artigo
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  14. 14
    Early growth response transcription factors: Key mediators of fibrosis and novel targets for anti-fibrotic therapy

    Early growth response transcription factors: Key mediators of fibrosis and novel targets for anti-fibrotic therapy mā Swati Bhattacharyya, Minghua Wu, Feng Fang, Warren G. Tourtellotte, Carol Feghali‐Bostwick, John Varga

    I whakaputaina 2011
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  15. 15
    A Peptide Derived from Endostatin Ameliorates Organ Fibrosis

    A Peptide Derived from Endostatin Ameliorates Organ Fibrosis mā Yukie Yamaguchi, Takahisa Takihara, Roger Chambers, Kristen L. Veraldi, Adriana T. Larregina, Carol Feghali‐Bostwick

    I whakaputaina 2012
    Whiwhi kuputuhi katoa
    Artigo
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  16. 16
    Sirtuin 3 Deregulation Promotes Pulmonary Fibrosis

    Sirtuin 3 Deregulation Promotes Pulmonary Fibrosis mā Meredith L. Sosulski, Rafael Góngora, Carol Feghali‐Bostwick, Joseph A. Lasky, Cecilia G. Sánchez

    I whakaputaina 2016
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  17. 17
    Insulin-like growth factor (IGF)-II- mediated fibrosis in pathogenic lung conditions

    Insulin-like growth factor (IGF)-II- mediated fibrosis in pathogenic lung conditions mā Sara M. Garrett, Eileen Hsu, Justin M. Thomas, Joseph M. Pilewski, Carol Feghali‐Bostwick

    I whakaputaina 2019
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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  18. 18
    Review: Interstitial Lung Disease Associated With Systemic Sclerosis and Idiopathic Pulmonary Fibrosis: How Similar and Distinct?

    Review: Interstitial Lung Disease Associated With Systemic Sclerosis and Idiopathic Pulmonary Fibrosis: How Similar and Distinct? mā Erica L. Herzog, Aditi Mathur, Andrew M. Tager, Carol Feghali‐Bostwick, Frank Schneider, John Varga

    I whakaputaina 2014
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  19. 19
    Localized expression of tenascin in systemic sclerosis–associated pulmonary fibrosis and its regulation by insulin‐like growth factor binding protein 3

    Localized expression of tenascin in systemic sclerosis–associated pulmonary fibrosis and its regulation by insulin‐like growth factor binding protein 3 mā Monique L. Brissett, Kristen L. Veraldi, Joseph M. Pilewski, Thomas A. Medsger, Carol Feghali‐Bostwick

    I whakaputaina 2011
    Whiwhi kuputuhi katoa
    Artigo
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  20. 20
    The K+ Channel KCa3.1 as a Novel Target for Idiopathic Pulmonary Fibrosis

    The K+ Channel KCa3.1 as a Novel Target for Idiopathic Pulmonary Fibrosis mā Katy M. Roach, S. Mark Duffy, William R. Coward, Carol Feghali‐Bostwick, Heike Wulff, Peter Bradding

    I whakaputaina 2013
    Whiwhi kuputuhi katoa Whiwhi kuputuhi katoa
    Artigo
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