Resultats de la cerca - Carol Feghali‐Bostwick

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  1. 1
    The Molecular Mechanisms of Systemic Sclerosis-Associated Lung Fibrosis

    The Molecular Mechanisms of Systemic Sclerosis-Associated Lung Fibrosis per Joe E. Mouawad, Carol Feghali‐Bostwick

    Publicat 2023
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  2. 2
    The Pro-Fibrotic Factor IGFBP-5 Induces Lung Fibroblast and Mononuclear Cell Migration

    The Pro-Fibrotic Factor IGFBP-5 Induces Lung Fibroblast and Mononuclear Cell Migration per Hidekata Yasuoka, Yukie Yamaguchi, Carol Feghali‐Bostwick

    Publicat 2009
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  3. 3
    The Mighty Fibroblast and Its Utility in Scleroderma Research

    The Mighty Fibroblast and Its Utility in Scleroderma Research per Sara M. Garrett, DeAnna Baker Frost, Carol Feghali‐Bostwick

    Publicat 2017
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  4. 4
    Interleukin‐13–producing CD8+ T cells mediate dermal fibrosis in patients with systemic sclerosis

    Interleukin‐13–producing CD8+ T cells mediate dermal fibrosis in patients with systemic sclerosis per Patrizia Fuschiotti, Adriana T. Larregina, Johnan Ho, Carol Feghali‐Bostwick, Thomas A. Medsger

    Publicat 2012
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  5. 5
    Human lung myofibroblast TGFβ1-dependent Smad2/3 signalling is Ca2+-dependent and regulated by KCa3.1 K+ channels

    Human lung myofibroblast TGFβ1-dependent Smad2/3 signalling is Ca2+-dependent and regulated by KCa3.1 K+ channels per Katy M. Roach, Carol Feghali‐Bostwick, Heike Wulff, Yassine Amrani, Peter Bradding

    Publicat 2015
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  6. 6
    Repression of IP-10 by Interactions between Histone Deacetylation and Hypermethylation in Idiopathic Pulmonary Fibrosis

    Repression of IP-10 by Interactions between Histone Deacetylation and Hypermethylation in Idiopathic Pulmonary Fibrosis per William R. Coward, Keira Watts, Carol Feghali‐Bostwick, Gisli Jenkins, Linhua Pang

    Publicat 2010
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  7. 7
    Increased constitutive αSMA and Smad2/3 expression in idiopathic pulmonary fibrosis myofibroblasts is KCa3.1-dependent

    Increased constitutive αSMA and Smad2/3 expression in idiopathic pulmonary fibrosis myofibroblasts is KCa3.1-dependent per Katy M. Roach, Heike Wulff, Carol Feghali‐Bostwick, Yassine Amrani, Peter Bradding

    Publicat 2014
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  8. 8
    IGFBP-5 Promotes Fibrosis via Increasing Its Own Expression and That of Other Pro-fibrotic Mediators

    IGFBP-5 Promotes Fibrosis via Increasing Its Own Expression and That of Other Pro-fibrotic Mediators per Xinh-Xinh Nguyen, Lutfiyya N. Muhammad, Paul J. Nietert, Carol Feghali‐Bostwick

    Publicat 2018
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  9. 9
    A central role for G9a and EZH2 in the epigenetic silencing of cyclooxygenase‐2 in idiopathic pulmonary fibrosis

    A central role for G9a and EZH2 in the epigenetic silencing of cyclooxygenase‐2 in idiopathic pulmonary fibrosis per William R. Coward, Carol Feghali‐Bostwick, Gisli Jenkins, Alan J. Knox, Linhua Pang

    Publicat 2014
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  10. 10
    Defective Histone Acetylation Is Responsible for the Diminished Expression of Cyclooxygenase 2 in Idiopathic Pulmonary Fibrosis

    Defective Histone Acetylation Is Responsible for the Diminished Expression of Cyclooxygenase 2 in Idiopathic Pulmonary Fibrosis per William R. Coward, Keiria Watts, Carol Feghali‐Bostwick, Alan J. Knox, Linhua Pang

    Publicat 2009
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  11. 11
    Gender differences in systemic sclerosis: relationship to clinical features, serologic status and outcomes

    Gender differences in systemic sclerosis: relationship to clinical features, serologic status and outcomes per Christine Peoples, Thomas A. Medsger, Mary Lucas, Bedda L. Rosario, Carol Feghali‐Bostwick

    Publicat 2016
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  12. 12
    Prominence of IL6, IGF, TLR, and Bioenergetics Pathway Perturbation in Lung Tissues of Scleroderma Patients With Pulmonary Fibrosis

    Prominence of IL6, IGF, TLR, and Bioenergetics Pathway Perturbation in Lung Tissues of Scleroderma Patients With Pulmonary Fibrosis per Ludivine Renaud, Willian A. da Silveira, Naoko Takamura, Gary Hardiman, Carol Feghali‐Bostwick

    Publicat 2020
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  13. 13
    Insulin-Like Growth Factor Binding Proteins 3 and 5 Are Overexpressed in Idiopathic Pulmonary Fibrosis and Contribute to Extracellular Matrix Deposition

    Insulin-Like Growth Factor Binding Proteins 3 and 5 Are Overexpressed in Idiopathic Pulmonary Fibrosis and Contribute to Extracellular Matrix Deposition per Joseph M. Pilewski, Lixin Liu, Adam C. Henry, Alycia V. Knauer, Carol Feghali‐Bostwick

    Publicat 2005
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  14. 14
    Early growth response transcription factors: Key mediators of fibrosis and novel targets for anti-fibrotic therapy

    Early growth response transcription factors: Key mediators of fibrosis and novel targets for anti-fibrotic therapy per Swati Bhattacharyya, Minghua Wu, Feng Fang, Warren G. Tourtellotte, Carol Feghali‐Bostwick, John Varga

    Publicat 2011
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  15. 15
    A Peptide Derived from Endostatin Ameliorates Organ Fibrosis

    A Peptide Derived from Endostatin Ameliorates Organ Fibrosis per Yukie Yamaguchi, Takahisa Takihara, Roger Chambers, Kristen L. Veraldi, Adriana T. Larregina, Carol Feghali‐Bostwick

    Publicat 2012
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  16. 16
    Sirtuin 3 Deregulation Promotes Pulmonary Fibrosis

    Sirtuin 3 Deregulation Promotes Pulmonary Fibrosis per Meredith L. Sosulski, Rafael Góngora, Carol Feghali‐Bostwick, Joseph A. Lasky, Cecilia G. Sánchez

    Publicat 2016
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  17. 17
    Insulin-like growth factor (IGF)-II- mediated fibrosis in pathogenic lung conditions

    Insulin-like growth factor (IGF)-II- mediated fibrosis in pathogenic lung conditions per Sara M. Garrett, Eileen Hsu, Justin M. Thomas, Joseph M. Pilewski, Carol Feghali‐Bostwick

    Publicat 2019
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  18. 18
    Review: Interstitial Lung Disease Associated With Systemic Sclerosis and Idiopathic Pulmonary Fibrosis: How Similar and Distinct?

    Review: Interstitial Lung Disease Associated With Systemic Sclerosis and Idiopathic Pulmonary Fibrosis: How Similar and Distinct? per Erica L. Herzog, Aditi Mathur, Andrew M. Tager, Carol Feghali‐Bostwick, Frank Schneider, John Varga

    Publicat 2014
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  19. 19
    Localized expression of tenascin in systemic sclerosis–associated pulmonary fibrosis and its regulation by insulin‐like growth factor binding protein 3

    Localized expression of tenascin in systemic sclerosis–associated pulmonary fibrosis and its regulation by insulin‐like growth factor binding protein 3 per Monique L. Brissett, Kristen L. Veraldi, Joseph M. Pilewski, Thomas A. Medsger, Carol Feghali‐Bostwick

    Publicat 2011
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  20. 20
    The K+ Channel KCa3.1 as a Novel Target for Idiopathic Pulmonary Fibrosis

    The K+ Channel KCa3.1 as a Novel Target for Idiopathic Pulmonary Fibrosis per Katy M. Roach, S. Mark Duffy, William R. Coward, Carol Feghali‐Bostwick, Heike Wulff, Peter Bradding

    Publicat 2013
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