Hakutulokset - Birgit Stallmeyer

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  1. 1
    How exome sequencing improves the diagnostics and management of men with non‐syndromic infertility

    How exome sequencing improves the diagnostics and management of men with non‐syndromic infertility Tekijä Birgit Stallmeyer, Ann‐Kristin Dicke, Frank Tüttelmann

    Julkaistu 2024
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    Revisão
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  2. 2
    Nitric oxide triggers enhanced induction of vascular endothelial growth factor expression in cultured keratinocytes (HaCaT) and during cutaneous wound repair

    Nitric oxide triggers enhanced induction of vascular endothelial growth factor expression in cultured keratinocytes (HaCaT) and during cutaneous wound repair Tekijä Stefan L. Frank, Birgit Stallmeyer, Heiko Kämpfer, Nicole Kolb, Josef Pfeilschifter

    Julkaistu 1999
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    Artigo
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  3. 3
    Large and Sustained Induction of Chemokines during Impaired Wound Healing in the Genetically Diabetic Mouse: Prolonged Persistence of Neutrophils and Macrophages during the Late Phase of Repair

    Large and Sustained Induction of Chemokines during Impaired Wound Healing in the Genetically Diabetic Mouse: Prolonged Persistence of Neutrophils and Macrophages during the Late Ph... Tekijä Christian Wetzler, Heiko Kämpfer, Birgit Stallmeyer, Josef Pfeilschifter, Stefan L. Frank

    Julkaistu 2000
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    Artigo
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  4. 4
    The Function of Nitric Oxide in Wound Repair: Inhibition of Inducible Nitric Oxide-Synthase Severely Impairs Wound Reepithelialization

    The Function of Nitric Oxide in Wound Repair: Inhibition of Inducible Nitric Oxide-Synthase Severely Impairs Wound Reepithelialization Tekijä Birgit Stallmeyer, Heiko Kämpfer, Nicole Kolb, Josef Pfeilschifter, Stefan L. Frank

    Julkaistu 1999
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    Artigo
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  5. 5
    Leptin enhances wound re-epithelialization and constitutes a direct function of leptin in skin repair

    Leptin enhances wound re-epithelialization and constitutes a direct function of leptin in skin repair Tekijä Stefan L. Frank, Birgit Stallmeyer, Heiko Kämpfer, Nicole Kolb, Josef Pfeilschifter

    Julkaistu 2000
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    Artigo
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  6. 6
    Molybdenum co‐factor biosynthesis: the <i>Arabidopsis thaliana</i> cDNA <i>cnx1</i> encodes a multifunctional two‐domain protein homologous to a mammalian neuroprotein, the insect protein Cinnamon and three <i>Escherichia coli</i> proteins

    Molybdenum co‐factor biosynthesis: the <i>Arabidopsis thaliana</i> cDNA <i>cnx1</i> encodes a multifunctional two‐domain protein homologous to a mammalian neuroprotein, the insect... Tekijä Birgit Stallmeyer, Michael Nerlich, Jochen Schiemann, Henner Brinkmann, Ralf R. Mendel

    Julkaistu 1995
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    Artigo
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  7. 7
    A Novel Keratinocyte Mitogen: Regulation of Leptin and its Functional Receptor in Skin Repair

    A Novel Keratinocyte Mitogen: Regulation of Leptin and its Functional Receptor in Skin Repair Tekijä Birgit Stallmeyer, Heiko Kämpfer, Josef Pfeilschifter, Stefan L. Frank, Maurizio Podda, Roland Kaufmann

    Julkaistu 2001
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    Artigo
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  8. 8
    T<sub>peak</sub>–T<sub>end</sub>interval and T<sub>peak</sub>–T<sub>end</sub>/QT ratio in patients with Brugada syndrome

    T<sub>peak</sub>–T<sub>end</sub>interval and T<sub>peak</sub>–T<sub>end</sub>/QT ratio in patients with Brugada syndrome Tekijä Sven Zumhagen, Eva M. Zeidler, Birgit Stallmeyer, Marko Ernsting, Lars Eckardt, Eric Schulze‐Bahr

    Julkaistu 2016
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    Artigo
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  9. 9
    Overlapping Cardiac Phenotype Associated with a Familial Mutation in the Voltage Sensor of the KCNQ1 Channel

    Overlapping Cardiac Phenotype Associated with a Familial Mutation in the Voltage Sensor of the KCNQ1 Channel Tekijä Ulrike Henrion, Sven Zumhagen, Katja Steinke, Nathalie Strutz‐Seebohm, Birgit Stallmeyer, Florian Läng, Eric Schulze‐Bahr, Guiscard Seebohm

    Julkaistu 2012
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    Artigo
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  10. 10
    Human iPS cell model of type 3 long QT syndrome recapitulates drug-based phenotype correction

    Human iPS cell model of type 3 long QT syndrome recapitulates drug-based phenotype correction Tekijä Daniela Malan, Miao Zhang, Birgit Stallmeyer, Jovanca Müller, Bernd K. Fleischmann, Eric Schulze‐Bahr, Philipp Sasse, Boris Greber

    Julkaistu 2016
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    Artigo
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  11. 11
    Gain‐of‐function mutation in <scp>TASK</scp>‐4 channels and severe cardiac conduction disorder

    Gain‐of‐function mutation in <scp>TASK</scp>‐4 channels and severe cardiac conduction disorder Tekijä Corinna Friedrich, Susanne Rinné, Sven Zumhagen, Aytuğ K. Kiper, Nicole Silbernagel, Michael F. Netter, Birgit Stallmeyer, Eric Schulze‐Bahr, Niels Decher

    Julkaistu 2014
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    Artigo
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  12. 12
    Impaired endocytosis of the ion channel TRPM4 is associated with human progressive familial heart block type I

    Impaired endocytosis of the ion channel TRPM4 is associated with human progressive familial heart block type I Tekijä Martin Kruse, Eric Schulze‐Bahr, Valerie A. Corfield, Alf Beckmann, Birgit Stallmeyer, Güven Kurtbay, Iris Ohmert, Ellen Schulze-Bahr, Paul A. Brink, Olaf Pongs

    Julkaistu 2009
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    Artigo
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  13. 13
    Familial Sinus Node Disease Caused by a Gain of GIRK (G-Protein Activated Inwardly Rectifying K <sup>+</sup> Channel) Channel Function

    Familial Sinus Node Disease Caused by a Gain of GIRK (G-Protein Activated Inwardly Rectifying K <sup>+</sup> Channel) Channel Function Tekijä Johanna Kuß, Birgit Stallmeyer, Matthias Goldstein, Susanne Rinné, Christiane Pees, Sven Zumhagen, Guiscard Seebohm, Niels Decher, Lutz Pott, Marie‐Cécile Kienitz, Eric Schulze‐Bahr

    Julkaistu 2019
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    Artigo
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  14. 14
    Mutational spectrum in the Ca2+-activated cation channel gene TRPM4 in patients with cardiac conductance disturbances

    Mutational spectrum in the Ca2+-activated cation channel gene TRPM4 in patients with cardiac conductance disturbances Tekijä Birgit Stallmeyer, Sven Zumhagen, Isabelle Denjoy, Guillaume Duthoit, Jean‐Louis Hébert, Xavier Ferrer, Svetlana Maugenre, Wilhelm Schmitz, Uwe Kirchhefer, Ellen Schulze-Bahr, Pascale Guicheney, Eric Schulze‐Bahr

    Julkaistu 2011
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    Artigo
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  15. 15
    A Mutation in the G-Protein Gene <i>GNB2</i> Causes Familial Sinus Node and Atrioventricular Conduction Dysfunction

    A Mutation in the G-Protein Gene <i>GNB2</i> Causes Familial Sinus Node and Atrioventricular Conduction Dysfunction Tekijä Birgit Stallmeyer, Johanna Kuß, S. Kotthoff, Sven Zumhagen, Kirsty Vowinkel, Susanne Rinné, Lina A. Matschke, Corinna Friedrich, Ellen Schulze-Bahr, Stephan Rust, Guiscard Seebohm, Niels Decher, Eric Schulze‐Bahr

    Julkaistu 2017
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    Artigo
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  16. 16
    DDX3Y is likely the key spermatogenic factor in the AZFa region that contributes to human non-obstructive azoospermia

    DDX3Y is likely the key spermatogenic factor in the AZFa region that contributes to human non-obstructive azoospermia Tekijä Ann‐Kristin Dicke, Adrian Pilatz, Margot J. Wyrwoll, Margus Punab, Christian Rückert, Liina Nagirnaja, Kenneth I. Aston, Donald F. Conrad, Sara Di Persio, Nina Neuhaus, Daniela Fietz, Maris Laan, Birgit Stallmeyer, Frank Tüttelmann

    Julkaistu 2023
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    Artigo
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  17. 17
    Sodium permeable and “hypersensitive” <scp>TREK</scp> ‐1 channels cause ventricular tachycardia

    Sodium permeable and “hypersensitive” <scp>TREK</scp> ‐1 channels cause ventricular tachycardia Tekijä Niels Decher, Beatriz Ortiz‐Bonnin, Corinna Friedrich, Marcus Schewe, Aytuğ K. Kiper, Susanne Rinné, Gunnar Seemann, Rémi Peyronnet, Sven Zumhagen, Daniel Bustos, Jens Kockskämper, Peter Köhl, Steffen Just, Wendy González, Thomas Baukrowitz, Birgit Stallmeyer, Eric Schulze‐Bahr

    Julkaistu 2017
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    Artigo
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  18. 18
    Estradiol regulates human QT-interval: acceleration of cardiac repolarization by enhanced KCNH2 membrane trafficking

    Estradiol regulates human QT-interval: acceleration of cardiac repolarization by enhanced KCNH2 membrane trafficking Tekijä Lars Anneken, Stefan Baumann, Patrick Vigneault, Péter Biliczki, Corinna Friedrich, Ling Xiao, Zenawit Girmatsion, Ina Takac, Ralf P. Brandes, Stefan Kissler, Inka Wiegratz, Sven Zumhagen, Birgit Stallmeyer, Stefan H. Hohnloser, Thomas Klingenheben, Eric Schulze‐Bahr, Stanley Nattel, Joachim R. Ehrlich

    Julkaistu 2015
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  19. 19
    Cardiac α-Actin ( <i>ACTC1</i> ) Gene Mutation Causes Atrial-Septal Defects Associated With Late-Onset Dilated Cardiomyopathy

    Cardiac α-Actin ( <i>ACTC1</i> ) Gene Mutation Causes Atrial-Septal Defects Associated With Late-Onset Dilated Cardiomyopathy Tekijä Derk Frank, Ashraf Yusuf Rangrez, Corinna Friedrich, Sven Dittmann, Birgit Stallmeyer, Pankaj Yadav, Alexander Bernt, Ellen Schulze-Bahr, Ankush Borlepawar, Wolfram‐Hubertus Zimmermann, Stefan Peischard, Guiscard Seebohm, Wolfgang A. Linke, Hideo A. Baba, Marcus Krüger, Andreas Unger, Philip Usinger, Norbert Frey, Eric Schulze‐Bahr

    Julkaistu 2019
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  20. 20
    Human fertilization in vivo and in vitro requires the CatSper channel to initiate sperm hyperactivation

    Human fertilization in vivo and in vitro requires the CatSper channel to initiate sperm hyperactivation Tekijä Samuel Young, Christian Schiffer, Alice Wagner, Jannika Patz, A. Potapenko, Leonie Herrmann, Verena Nordhoff, Tim Pock, Claudia Krallmann, Birgit Stallmeyer, Albrecht Röpke, Michelina Kierzek, Cristina Biagioni, Tao Wang, Lars Haalck, Dirk Deuster, Jan N. Hansen, Dagmar Wachten, Benjamin Risse, Hermann M. Behre, Stefan Schlatt, Sabine Kliesch, Frank Tüttelmann, Christoph Brenker, Timo Strünker

    Julkaistu 2024
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    Artigo
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