Resultados da busca - Alberto Serrano‐Pozo

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  1. 1
    Is Alzheimer’s Disease Risk Modifiable?

    Is Alzheimer’s Disease Risk Modifiable? por Alberto Serrano‐Pozo, John H. Growdon

    Publicado em 2019
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  2. 2
    Deciphering the Astrocyte Reaction in Alzheimer’s Disease

    Deciphering the Astrocyte Reaction in Alzheimer’s Disease por Beatriz Gomez Perez‐Nievas, Alberto Serrano‐Pozo

    Publicado em 2018
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  3. 3
    APOE and Alzheimer's disease: advances in genetics, pathophysiology, and therapeutic approaches

    APOE and Alzheimer's disease: advances in genetics, pathophysiology, and therapeutic approaches por Alberto Serrano‐Pozo, Sudeshna Das, Bradley T. Hyman

    Publicado em 2020
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  4. 4
    Four Decades of Research in Alzheimer’s Disease (1975–2014): A Bibliometric and Scientometric Analysis

    Four Decades of Research in Alzheimer’s Disease (1975–2014): A Bibliometric and Scientometric Analysis por Alberto Serrano‐Pozo, Georgina M. Aldridge, Qiang Zhang

    Publicado em 2017
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  5. 5
    Neuropathological Alterations in Alzheimer Disease

    Neuropathological Alterations in Alzheimer Disease por Alberto Serrano‐Pozo, Matthew P. Frosch, Eliezer Masliah, B. T. Hyman

    Publicado em 2011
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  6. 6
    Association of <i>APOE</i> Genotype With Heterogeneity of Cognitive Decline Rate in Alzheimer Disease

    Association of <i>APOE</i> Genotype With Heterogeneity of Cognitive Decline Rate in Alzheimer Disease por Jing Qian, Rebecca A. Betensky, Bradley T. Hyman, Alberto Serrano‐Pozo

    Publicado em 2021
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  7. 7
    Plaque-Associated Local Toxicity Increases over the Clinical Course of Alzheimer Disease

    Plaque-Associated Local Toxicity Increases over the Clinical Course of Alzheimer Disease por Alberto Serrano‐Pozo, Rebecca A. Betensky, Matthew P. Frosch, Bradley T. Hyman

    Publicado em 2015
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  8. 8
    Meta-analysis of mouse transcriptomic studies supports a context-dependent astrocyte reaction in acute CNS injury versus neurodegeneration

    Meta-analysis of mouse transcriptomic studies supports a context-dependent astrocyte reaction in acute CNS injury versus neurodegeneration por Sudeshna Das, Zhaozhi Li, Ayush Noori, Bradley T. Hyman, Alberto Serrano‐Pozo

    Publicado em 2020
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  9. 9
    Characterization of the 18 kDa translocator protein (TSPO) expression in <i>post‐mortem</i> normal and Alzheimer's disease brains

    Characterization of the 18 kDa translocator protein (TSPO) expression in <i>post‐mortem</i> normal and Alzheimer's disease brains por Yaxing Gui, Jordan D. Marks, Sudeshna Das, Bradley T. Hyman, Alberto Serrano‐Pozo

    Publicado em 2019
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  10. 10
    Systematic review and meta-analysis of human transcriptomics reveals neuroinflammation, deficient energy metabolism, and proteostasis failure across neurodegeneration

    Systematic review and meta-analysis of human transcriptomics reveals neuroinflammation, deficient energy metabolism, and proteostasis failure across neurodegeneration por Ayush Noori, Aziz M. Mezlini, Bradley T. Hyman, Alberto Serrano‐Pozo, Sudeshna Das

    Publicado em 2020
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  11. 11
    <scp><i>APOE</i></scp>ε2 is associated with milder clinical and pathological <scp>A</scp>lzheimer disease

    <scp><i>APOE</i></scp>ε2 is associated with milder clinical and pathological <scp>A</scp>lzheimer disease por Alberto Serrano‐Pozo, Jing Qian, Sarah E. Monsell, Rebecca A. Betensky, Bradley T. Hyman

    Publicado em 2015
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  12. 12
    Systematic review of human post‐mortem immunohistochemical studies and bioinformatics analyses unveil the complexity of astrocyte reaction in Alzheimer's disease

    Systematic review of human post‐mortem immunohistochemical studies and bioinformatics analyses unveil the complexity of astrocyte reaction in Alzheimer's disease por Lucía Viejo, Ayush Noori, Emily Merrill, Sudeshna Das, Bradley T. Hyman, Alberto Serrano‐Pozo

    Publicado em 2021
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  13. 13
    A Phenotypic Change But Not Proliferation Underlies Glial Responses in Alzheimer Disease

    A Phenotypic Change But Not Proliferation Underlies Glial Responses in Alzheimer Disease por Alberto Serrano‐Pozo, Teresa Gómez‐Isla, John H. Growdon, Matthew P. Frosch, Bradley T. Hyman

    Publicado em 2013
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  14. 14
    Characterization of monoamine oxidase-B (MAO-B) as a biomarker of reactive astrogliosis in Alzheimer’s disease and related dementias

    Characterization of monoamine oxidase-B (MAO-B) as a biomarker of reactive astrogliosis in Alzheimer’s disease and related dementias por Methasit Jaisa-aad, Clara Muñoz‐Castro, Molly A Healey, Bradley T. Hyman, Alberto Serrano‐Pozo

    Publicado em 2024
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  15. 15
    Mitochondrial Alterations near Amyloid Plaques in an Alzheimer's Disease Mouse Model

    Mitochondrial Alterations near Amyloid Plaques in an Alzheimer's Disease Mouse Model por Hong Xie, Ji‐Song Guan, Laura A. Borrelli, Jing Xu, Alberto Serrano‐Pozo, Brian J. Bacskai

    Publicado em 2013
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  16. 16
    Examination of the Clinicopathologic Continuum of Alzheimer Disease in the Autopsy Cohort of the National Alzheimer Coordinating Center

    Examination of the Clinicopathologic Continuum of Alzheimer Disease in the Autopsy Cohort of the National Alzheimer Coordinating Center por Alberto Serrano‐Pozo, Jing Qian, Sarah E. Monsell, Matthew P. Frosch, Rebecca A. Betensky, Bradley T. Hyman

    Publicado em 2013
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  17. 17
    Astrocyte tau deposition in progressive supranuclear palsy is associated with dysregulation of MAPT transcription

    Astrocyte tau deposition in progressive supranuclear palsy is associated with dysregulation of MAPT transcription por Rosemary J. Jackson, Alexandra Melloni, Dustin P. Fykstra, Alberto Serrano‐Pozo, Leslie A. Shinobu, Bradley T. Hyman

    Publicado em 2024
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  18. 18
    Presenilin-1 adopts pathogenic conformation in normal aging and in sporadic Alzheimer’s disease

    Presenilin-1 adopts pathogenic conformation in normal aging and in sporadic Alzheimer’s disease por Lara Wahlster, Muriel Arimon, Navine Nasser‐Ghodsi, Kathryn Leigh Post, Alberto Serrano‐Pozo, Kengo Uemura, Oksana Berezovska

    Publicado em 2012
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  19. 19
    The Synaptic Accumulation of Hyperphosphorylated Tau Oligomers in Alzheimer Disease Is Associated With Dysfunction of the Ubiquitin-Proteasome System

    The Synaptic Accumulation of Hyperphosphorylated Tau Oligomers in Alzheimer Disease Is Associated With Dysfunction of the Ubiquitin-Proteasome System por Hwan‐Ching Tai, Alberto Serrano‐Pozo, Tadafumi Hashimoto, Matthew P. Frosch, Tara L. Spires‐Jones, Bradley T. Hyman

    Publicado em 2012
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  20. 20
    Frequent and symmetric deposition of misfolded tau oligomers within presynaptic and postsynaptic terminals in Alzheimer’s disease

    Frequent and symmetric deposition of misfolded tau oligomers within presynaptic and postsynaptic terminals in Alzheimer’s disease por Hwan‐Ching Tai, Bo Y. Wang, Alberto Serrano‐Pozo, Matthew P. Frosch, Tara L. Spires‐Jones, Bradley T. Hyman

    Publicado em 2014
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