Результаты поиска - Alan Permutt

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  1. 1
    Effect of Glucose on Initiation and Elongation Rates in Isolated Rat Pancreatic Islets

    Effect of Glucose on Initiation and Elongation Rates in Isolated Rat Pancreatic Islets по M. Alan Permutt

    Опубликовано 1974
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  2. 2
    Insulin Biosynthesis

    Insulin Biosynthesis по M. Alan Permutt, David Kipnis

    Опубликовано 1972
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    Artigo
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  3. 3
    Evidence for increased recombination near the human insulin gene: implication for disease association studies.

    Evidence for increased recombination near the human insulin gene: implication for disease association studies. по Aravinda Chakravarti, Steven C. Elbein, M. Alan Permutt

    Опубликовано 1986
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  4. 4
    Amino acid sequence of catfish pancreatic somatostatin I.

    Amino acid sequence of catfish pancreatic somatostatin I. по Hideki Oyama, Ralph Bradshaw, Owen J. Bates, Alan Permutt

    Опубликовано 1980
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  5. 5
    Evaluation of rat insulin messenger RNA in pancreatic and extrapancreatic tissues

    Evaluation of rat insulin messenger RNA in pancreatic and extrapancreatic tissues по Stephen J. Giddings, John M. Chirgwin, M. Alan Permutt

    Опубликовано 1985
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  6. 6
    Diabetes and Insulin Secretion

    Diabetes and Insulin Secretion по Joseph C. Koster, M. Alan Permutt, Colin G. Nichols

    Опубликовано 2005
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  7. 7
    Glucose transporter levels in spontaneously obese (db/db) insulin-resistant mice.

    Glucose transporter levels in spontaneously obese (db/db) insulin-resistant mice. по László Korányi, David E. James, Mike Mueckler, M. Alan Permutt

    Опубликовано 1990
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  8. 8
    Mice with beta cell overexpression of glycogen synthase kinase-3β have reduced beta cell mass and proliferation

    Mice with beta cell overexpression of glycogen synthase kinase-3β have reduced beta cell mass and proliferation по Z. Liu, Katsuya Tanabe, Ernesto Bernal–Mizrachi, M. Alan Permutt

    Опубликовано 2008
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  9. 9
    Presenilins, Notch dose control the fate of pancreatic endocrine progenitors during a narrow developmental window

    Presenilins, Notch dose control the fate of pancreatic endocrine progenitors during a narrow developmental window по Corentin Cras‐Méneur, Lin Li, Raphael Kopan, M. Alan Permutt

    Опубликовано 2009
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  10. 10
    Glucose Regulates Foxo1 Through Insulin Receptor Signaling in the Pancreatic Islet β-cell

    Glucose Regulates Foxo1 Through Insulin Receptor Signaling in the Pancreatic Islet β-cell по Sara C. Martinez, Corentin Cras‐Méneur, Ernesto Bernal–Mizrachi, M. Alan Permutt

    Опубликовано 2006
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  11. 11
    Islet β cell expression of constitutively active Akt1/PKBα induces striking hypertrophy, hyperplasia, and hyperinsulinemia

    Islet β cell expression of constitutively active Akt1/PKBα induces striking hypertrophy, hyperplasia, and hyperinsulinemia по Ernesto Bernal–Mizrachi, Wen Wu, Sarah Stahlhut, Cris M. Welling, M. Alan Permutt

    Опубликовано 2001
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  12. 12
    Effects of altered glucose homeostasis on glucose transporter expression in skeletal muscle of the rat.

    Effects of altered glucose homeostasis on glucose transporter expression in skeletal muscle of the rat. по R. E. Bourey, László Korányi, David E. James, Mike Mueckler, M. Alan Permutt

    Опубликовано 1990
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  13. 13
    Islet β cell expression of constitutively active Akt1/PKBα induces striking hypertrophy, hyperplasia, and hyperinsulinemia

    Islet β cell expression of constitutively active Akt1/PKBα induces striking hypertrophy, hyperplasia, and hyperinsulinemia по Ernesto Bernal–Mizrachi, Wen Wu, Sarah Stahlhut, Cris M. Welling, M. Alan Permutt

    Опубликовано 2001
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  14. 14
    Endoplasmic Reticulum Stress–Induced Apoptosis Is Partly Mediated by Reduced Insulin Signaling Through Phosphatidylinositol 3-Kinase/Akt and Increased Glycogen Synthase Kinase-3β in Mouse Insulinoma Cells

    Endoplasmic Reticulum Stress–Induced Apoptosis Is Partly Mediated by Reduced Insulin Signaling Through Phosphatidylinositol 3-Kinase/Akt and Increased Glycogen Synthase Kinase-3β i... по Shanthi Srinivasan, Mitsuru Ohsugi, Zhonghao Liu, Szabolcs Fátrai, Ernesto Bernal–Mizrachi, M. Alan Permutt

    Опубликовано 2005
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  15. 15
    A mutation in the Glut2 glucose transporter gene of a diabetic patient abolishes transport activity.

    A mutation in the Glut2 glucose transporter gene of a diabetic patient abolishes transport activity. по Mike Mueckler, Michael Kruse, M. Strube, A. C. Riggs, Ken C. Chiu, M. Alan Permutt

    Опубликовано 1994
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  16. 16
    Cloning and functional expression of a human pancreatic islet glucose-transporter cDNA.

    Cloning and functional expression of a human pancreatic islet glucose-transporter cDNA. по M. Alan Permutt, László Korányi, K. Keller, Paul E. Lacy, D W Scharp, Mike Mueckler

    Опубликовано 1989
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  17. 17
    Missense mutations in the pancreatic islet beta cell inwardly rectifying K + channel gene (KIR6.2/BIR ): a meta-analysis suggests a role in the polygenic basis of Type II diabetes mellitus in Caucasians

    Missense mutations in the pancreatic islet beta cell inwardly rectifying K + channel gene (KIR6.2/BIR ): a meta-analysis suggests a role in the polygenic basis of Type II diabetes... по El Habib Hani, Philippe Boutin, Éric Durand, Hiroshi Inoue, M. Alan Permutt, Gilberto Velho, Philippe Froguel

    Опубликовано 1998
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  18. 18
    Diabetes Mellitus and Optic Atrophy: A Study of Wolfram Syndrome in the Lebanese Population

    Diabetes Mellitus and Optic Atrophy: A Study of Wolfram Syndrome in the Lebanese Population по R. Medlej, J. Wasson, Patrick Baz, Sami T. Azar, Ibrahim Salti, Jacques Loiselet, Alan Permutt, G Halaby

    Опубликовано 2004
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  19. 19
    Wolframin Expression Induces Novel Ion Channel Activity in Endoplasmic Reticulum Membranes and Increases Intracellular Calcium

    Wolframin Expression Induces Novel Ion Channel Activity in Endoplasmic Reticulum Membranes and Increases Intracellular Calcium по Abdullah A. Osman, Mitsuyoshi Saito, Carol M. Makepeace, M. Alan Permutt, Paul H. Schlesinger, Mike Mueckler

    Опубликовано 2003
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  20. 20
    Conditional ablation of Gsk-3β in islet beta cells results in expanded mass and resistance to fat feeding-induced diabetes in mice

    Conditional ablation of Gsk-3β in islet beta cells results in expanded mass and resistance to fat feeding-induced diabetes in mice по Y. Liu, Katsuya Tanabe, Delphine Baronnier, Satish Patel, James R. Woodgett, Corentin Cras‐Méneur, M. Alan Permutt

    Опубликовано 2010
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