檢索結果 - Alam, Hunain

Novel function of keratins 5 and 14 in proliferation and differentiation of stratified epithelial cells 由 Alam, Hunain, Sehgal, Lalit, Kundu, Samrat T., Dalal, Sorab N., Vaidya, Milind M.

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Transcriptional Repression of Histone Deacetylase 3 by the Histone Demethylase KDM2A Is Coupled to Tumorigenicity of Lung Cancer Cells 由 Dhar, Shilpa S., Alam, Hunain, Li, Na, Wagner, Klaus W., Chung, Jimyung, Ahn, Yeo Won, Lee, Min Gyu

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HP1γ promotes lung adenocarcinoma by downregulating the transcription-repressive regulators NCOR2 and ZBTB7A 由 Alam, Hunain, Li, Na, Dhar, Shilpa S., Wu, Sarah J., Lv, Jie, Chen, Kaifu, Flores, Elsa R., Baseler, Laura, Lee, Min Gyu

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Understanding the Role of Keratins 8 and 18 in Neoplastic Potential of Breast Cancer Derived Cell Lines 由 Iyer, Sapna V., Dange, Prerana P., Alam, Hunain, Sawant, Sharada S., Ingle, Arvind D., Borges, Anita M., Shirsat, Neelam V., Dalal, Sorab N., Vaidya, Milind M.

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Plakophilin3 Loss Leads to an Increase in PRL3 Levels Promoting K8 Dephosphorylation, Which Is Required for Transformation and Metastasis 由 Khapare, Nileema, Kundu, Samrat T., Sehgal, Lalit, Sawant, Mugdha, Priya, Rashmi, Gosavi, Prajakta, Gupta, Neha, Alam, Hunain, Karkhanis, Madhura, Naik, Nishigandha, Vaidya, Milind M., Dalal, Sorab N.

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Loss of Keratin 8 Phosphorylation Leads to Increased Tumor Progression and Correlates with Clinico-Pathological Parameters of OSCC Patients 由 Alam, Hunain, Gangadaran, Prakash, Bhate, Amruta V., Chaukar, Devendra A., Sawant, Sharada S., Tiwari, Richa, Bobade, Jyoti, Kannan, Sadhana, D'cruz, Anil K., Kane, Shubhada, Vaidya, Milind M.

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Vimentin regulates differentiation switch via modulation of keratin 14 levels and their expression together correlates with poor prognosis in oral cancer patients 由 Dmello, Crismita, Sawant, Sharada, Alam, Hunain, Gangadaran, Prakash, Mogre, Saie, Tiwari, Richa, D’Souza, Zinia, Narkar, Manish, Thorat, Rahul, Patil, Komal, Chaukar, Devendra, Kane, Shubhada, Vaidya, Milind

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Fascin overexpression promotes neoplastic progression in oral squamous cell carcinoma 由 Alam, Hunain, Bhate, Amruta V, Gangadaran, Prakash, Sawant, Sharda S, Salot, Shimul, Sehgal, Lalit, Dange, Prerana P, Chaukar, Devendra A, D'cruz, Anil K, Kannanl, Sadhna, Gude, Rajiv, Kane, Shubhada, Dalal, Sorab N, Vaidya, Milind M

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MLL4 Is Required to Maintain Broad H3K4me3 Peaks and Super-enhancers at Tumor Suppressor Genes 由 Dhar, Shilpa S., Zhao, Dongyu, Lin, Tao, Gu, Bingnan, Pal, Khusboo, Wu, Sarah J., Alam, Hunain, Lv, Jie, Yun, Kyuson, Gopalakrishnan, Vidya, Flores, Elsa R., Northcott, Paul A., Rajaram, Veena, Li, Wei, Shilatifard, Ali, Sillitoe, Roy V., Chen, Kaifu, Lee, Min Gyu

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KDM2A promotes lung tumorigenesis by epigenetically enhancing ERK1/2 signaling 由 Wagner, Klaus W., Alam, Hunain, Dhar, Shilpa S., Giri, Uma, Li, Na, Wei, Yongkun, Giri, Dipak, Cascone, Tina, Kim, Jae-Hwan, Ye, Yuanqing, Multani, Asha S., Chan, Chia-Hsin, Erez, Baruch, Saigal, Babita, Chung, Jimyung, Lin, Hui-Kuan, Wu, Xifeng, Hung, Mien-Chie, Heymach, John V., Lee, Min Gyu

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KMT2D deficiency impairs super-enhancers to confer a glycolytic vulnerability in lung cancer 由 Alam, Hunain, Tang, Ming, Maitituoheti, Mayinuer, Dhar, Shilpa S., Kumar, Manish, Han, Chae Young, Ambati, Chandra Shekar R, Amin, Samir B., Gu, Bingnan, Chen, Tsai-Yu, Lin, Yu-Hsi, Chen, Jichao, Muller, Florian L., Putluri, Nagireddy, Flores, Elsa R., DeMayo, Francesco J., Baseler, Laura, Rai, Kunal, Lee, Min Gyu

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Enhancer Reprogramming Confers Dependence on Glycolysis and IGF Signaling in KMT2D Mutant Melanoma 由 Maitituoheti, Mayinuer, Keung, Emily Z., Tang, Ming, Yan, Liang, Alam, Hunain, Han, Guangchun, Singh, Anand K., Raman, Ayush T., Terranova, Christopher, Sarkar, Sharmistha, Orouji, Elias, Amin, Samir B., Sharma, Sneha, Williams, Maura, Samant, Neha S., Dhamdhere, Mayura, Zheng, Norman, Shah, Tara, Shah, Amiksha, Axelrad, Jacob B., Anvar, Nazanin E., Lin, Yu-Hsi, Jiang, Shan, Chang, Edward Q., Ingram, Davis R., Wang, Wei-Lien, Lazar, Alexander, Lee, Min Gyu, Muller, Florian, Wang, Linghua, Ying, Haoqiang, Rai, Kunal

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CCNE1 amplification is synthetic lethal with PKMYT1 kinase inhibition 由 Gallo, David, Young, Jordan T. F., Fourtounis, Jimmy, Martino, Giovanni, Álvarez-Quilón, Alejandro, Bernier, Cynthia, Duffy, Nicole M., Papp, Robert, Roulston, Anne, Stocco, Rino, Szychowski, Janek, Veloso, Artur, Alam, Hunain, Baruah, Prasamit S., Fortin, Alexanne Bonneau, Bowlan, Julian, Chaudhary, Natasha, Desjardins, Jessica, Dietrich, Evelyne, Fournier, Sara, Fugère-Desjardins, Chloe, Goullet de Rugy, Theo, Leclaire, Marie-Eve, Liu, Bingcan, Bhaskaran, Vivek, Mamane, Yael, Melo, Henrique, Nicolas, Olivier, Singhania, Akul, Szilard, Rachel K., Tkáč, Ján, Yin, Shou Yun, Morris, Stephen J., Zinda, Michael, Marshall, C. Gary, Durocher, Daniel

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